Fruit flies normally produce two types of antimicrobial peptides (defensin and drosomycin). Mutant fruit flies were generated that produced no antimicrobial peptides, defensin only, or drosomycin only. Wild-type flies and the mutant flies were then tested for survival after infection with Micrococcus luteus bacteria.

What statement about these data is true?

The two kinds of antimicrobial peptides (defensin and drosomycin) improve as a results of two central genes C and P within the wild kind. Within the deficiency of factor C or P or each, there's no development of antimicrobial peptides within the mutant kind. The development of antimicrobial peptides is that the collective results of the organic chemistry responses of cistron C and P. thus mutant varieties with solely defensin (produce of cistron C) or drosomycin (produce of gene P) peptides, or no peptides once infested with fungus genus crassa yeasts, doesn't endure although the wild kind continued.

This unit evidences that the two prevailing genetic factor C and P (genetic factor for antimicrobial peptides) are strongly connected with one another and as there's no biological process happened among these two genetic factor there is no discrimination, thence solely maternal amalgamation are articulated. So, the wild kind fruit flies can ready to endure once verified with the fungi. This can be additionally AN example of regressive transformation.